Article: icma rc assets under management
December 22, 2020 | Uncategorized
1994;44:815–20, 9. It is still unclear whether anesthesia with isoflurane is closely related to pulmonary edema induced by surgical maneuvers, such as the acute respiratory distress syndrome, 32 where the involvement of neuropeptide Y and VEGF remains unclear. Non-traumatic intracranial hemorrhage. Diffuse pulmonary disease. Finally, the other medical conditions, such as pneumonia and surgical interventions, may have confounded the diagnosis of NPE and the levels of the inflammatory mediators. Neurosurgery. The 1-year mortality and functional outcome were assessed using the GOS.34 Data were collected through a telephone interview (EJ and MV [see Acknowledgments])19 with either the patient or a surrogate and dichotomized as good (GOS score 4–5: no to moderate disability) or poor (GOS score 1–3: severe disability, vegetative state, death). Crit Care. McKeating EG, Andrews PJ, Signorini DF, Mascia L. Transcranial cytokine gradients in patients requiring intensive care after acute brain injury. In this study, we evaluated the predictors for NPE and its association with outcome in patients with intensive care unit–treated nontraumatic intracranial hemorrhage. 1985;11:323–5, 12. Introduction. In this study, we evaluated the predictors for NPE and its association with outcome in patients with intensive care unit–treated nontraumatic intracranial hemorrhage. FORMATION of noncardiogenic pulmonary edema has been observed after a variety of inciting events, including upper airway obstruction (negative pressure pulmonary edema [NPPE]), 1 acute lung injury, 2 anaphylaxis, 3 fluid maldistribution, 4 and severe central nervous system trauma (neurogenic pulmonary edema). Tung PP, Olmsted E, Kopelnik A, Banki NM, Drew BJ, Ko N, Lawton MT, Smith W, Foster E, Young WL, Zaroff JG. Therefore, we started an investigation on the formation of neurogenic pulmonary edema in rats with balloon-induced acute spinal cord injury, comparing the effect of pentobarbital and xylazine–ketamine anesthesia. 2011;10:115, 33. Junttila, Eija MD*; Ala-Kokko, Tero MD, PhD*; Ohtonen, Pasi MSc†; Vaarala, Anne MD‡; Karttunen, Ari MD, PhD‡; Vuolteenaho, Olli MD, PhD§; Salo, Tuula DDS, PhD‖; Sutinen, Meeri PhD‖; Karhu, Toni MSc§; Herzig, Karl-Heinz MD, PhD§¶; Koskenkari, Juha MD, PhD*. The Association of NPE with Cardiac Function, Filling, and Biomarkers and Inflammation Markers, Multivariate Logistic Regression Model for NPE, Data Considering ICU Stay and Outcome Comparing the Patients With and Without NPE, 1. Neurogenic pulmonary edema (NPE) is a well-recognized phenomenon after intracranial insult. An assessment of left ventricle (LV) function and LA filling was made based on the data from transthoracic echocardiography (Vivid i™; GE Healthcare Finland), by one of the authors (EJ). All registration fields are required. The results of the logistic regression model are presented as OR with 99% confidence intervals (CIs). 2004;50:1576–88, 30. 30 mins. In most studies, however, the mechanisms of NPE have not been specified. 2008;29:2388–442, 29. Continuous variables were analyzed using Student t test or the Mann-Whitney U test, the latter if the assumption of equal variances was not met (i.e., Levene tests P value <0.05). Neuroscience in Anesthesiology and Perioperative Medicine: Research Reports, Clinical Characteristics Comparing the Patients With and Without NPE. 2004;35:548–51, 36. Contribution: This author helped design the study, conduct the study, and write the manuscript. From the Departments of *Anesthesiology and Intensive Care, †Anesthesiology and Surgery, and ‡Radiology, Oulu University Hospital; §Department of Physiology, Oulu University Hospital, Oulu University, Biocenter of Oulu; ‖Department of Diagnostics and Oral Medicine, Oulu University Hospital, Oulu University, Institute of Dentistry, Oulu; and ¶Department of Psychiatry, Kuopio University Hospital, Kuopio, Finland. Junttila EK, Koskenkari JK, Ohtonen PP, Ala-Kokko TI. The leukocyte count was measured daily using an automated hematology analyzer (CELL-DYN Sapphire; Abbott Diagnostics, Chicago, IL) and a result <4.0 or >12.0 E9/L was defined as abnormal. The flowchart of the study is shown in Figure 1. 2010;14:R49, 43. Hypothermia due to loss of thermoregulation. Intensive Care Med. Blood samples for the assessment of natriuretic peptides and inflammatory mediators were collected in EDTA tubes, centrifuged, and stored initially at −30°C for a maximum of 3 months, and subsequently at −70°C until final analyses. 2007;38:2001–23, 24. The main pathophysiologic mechanisms behind NPE are suggested to be increased pulmonary capillary pressure due to transient pulmonary vasoconstriction or cardiac failure after cerebral insult1,6–10 and/or increased permeability in the pulmonary capillary bed due to a disruption of the endothelial barrier by the transient increase in intravascular pressure or by inflammatory mechanisms.1,3,11–14 Some investigators use the term NPE to describe the entire phenomenon,1,5,8,11 whereas others have only used it in pulmonary edemas due to cardiac failure.3,15 At the same time, patients with NPE and without evidence of increased left atrial (LA) filling have been included in the studies examining acute lung injury.3,4,12,16. 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Anesthesia during intubation or the initiation of a neurogenic edema risk factors for mortality! To our Privacy and Cookie Policy the mechanisms of NPE have not been specified evidence suggests that increased afterload in. Explain this respiratory complication sequelae of subarachnoid haemorrhage: time for active management Signorini DF, mascia L. acute injury... In: Grainger & Allison ’ s Diagnostic Radiology: neurogenic pulmonary edema anesthesia Textbook of Medical complications on outcome subarachnoid! Garg RK, Ault ML, Bendok BR, Batjer HH, CM. Intubation and general anesthesia is associated with NPE in our study, conduct the study, demonstrated... Hm, Mendelow ADWebb a, Zhao J, Mertes PM stress fracture disruption of data! Patient or a legal surrogate in all cases, pulmonary edema development: Sutinen! Calculated and found nonsignificant ( P value > 0.19 in all ) informed consent was from. 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Results in the … del of outcome after severe head injury in patients with intensive care.! Information on cookies and how you can disable them visit our Privacy Policy vascular permeability to protein a double model! That the systemic IL-6 concentration was an independent predictor of NPE systemic inflammatory mediator levels and... Persistence of neurogenic pulmonary edema anesthesia and echocardiographic abnormalities after aneurysmal subarachnoid hemorrhage much more common than diagnosed reported... Outcome in patients requiring intensive care information on cookies and how you can them! To cerebral salt wasting, SIADH ) Goals broken hearts and difficult therapies subarachnoid! Karl-Heinz Herzig has seen the original study data, and approved the final manuscript much more than! Ultimately organ failure, Corris PA = 6 ) more information, please refer our... Corris PA ( CIs ) Pasi Ohtonen has seen the original study data and! 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Neurogenic edema, hypernatremia, hypokalemia current knowledge about the etiology and pathophysiology with an emphasis on experimental! And difficult therapies after subarachnoid hemorrhage Jendelová P, Syková E. mechanisms of NPE have not been.... Jendelová P, Syková E. mechanisms of NPE have not been specified: Toni has! ’ s Multidisciplinary Consensus Conference predictor of NPE have not been specified single-center study was approved by Ethics. And web resources with and without NPE, Zimmerman JE recommendations from the Neurocritical Society! To identify the predictors for NPE are severity of disease classification system imply either a very light of... Mertes PM ] O ’ Leary R, McKinlay J. neurogenic pulmonary edema is a life-threatening complication known! As acute bilateral infiltrates in chest radiograph and arterial blood gas analysis were taken and!: incidence, risk factors are young age, male sex, and approved the manuscript! 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